A research team from Jackson Laboratory has found that the misfolded proteins implicated in several cardiac diseases could be the result of mistranslations during the "editing" process of protein synthesis rather than a mutated gene. In 2006, the laboratory JAX Professor and Howard Hughes Medical Investigator, Susan Ackerman, Ph.D., showed that the movement disorders in a mouse model with a mutation called sti (for "sticky," referring to the appearance of the animal's ...
via Medindia Health News
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