A new study funded by the National Institutes of Health reveals that removing toxic debris that accumulates in patients' brains by blocking activity of a regulator protein called CD33 could be a potential strategy in stemming the process involved in Alzheimer's disease. "Too much CD33 activity appears to promote late-onset Alzheimer's by preventing support cells from clearing out toxic plaques, key risk factors for the disease," explained Rudolph Tanzi, Ph.D., of ...Sunday 28 April 2013
Suppressing Protein Regulator Activity can Stem Alzheimer's Disease Process
A new study funded by the National Institutes of Health reveals that removing toxic debris that accumulates in patients' brains by blocking activity of a regulator protein called CD33 could be a potential strategy in stemming the process involved in Alzheimer's disease. "Too much CD33 activity appears to promote late-onset Alzheimer's by preventing support cells from clearing out toxic plaques, key risk factors for the disease," explained Rudolph Tanzi, Ph.D., of ...
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